Functional disruption of the Golgi apparatus protein ARF1 sensitizes MDA-MB-231 breast cancer cells to the antitumor drugs Actinomycin D and Vinblastine through ERK and AKT signaling

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dc.contributor.authorLuchsinger, Charlotte
dc.contributor.authorAguilar, Marcelo
dc.contributor.authorBurgos, Patricia V.
dc.contributor.authorEhrenfeld, Pamela
dc.contributor.authorMardones Cofré, Gonzalo Antonio
dc.date.accessioned2020-11-25T17:31:31Z
dc.date.available2020-11-25T17:31:31Z
dc.date.issued2018
dc.fuente.origenConveris
dc.identifier.doi10.1371/journal.pone.0195401
dc.identifier.issn1932-6203
dc.identifier.urihttps://doi.org/10.1371/journal.pone.0195401
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/48580
dc.issue.numeroNo. 4
dc.language.isoen
dc.pagina.final25
dc.pagina.inicio1
dc.revistaPLoS ONEes_ES
dc.rightsacceso restringido
dc.titleFunctional disruption of the Golgi apparatus protein ARF1 sensitizes MDA-MB-231 breast cancer cells to the antitumor drugs Actinomycin D and Vinblastine through ERK and AKT signalinges_ES
dc.typeartículo
dc.volumenVol. 13
sipa.codpersvinculados6969
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